Clinical role of 18F-FDG PET/CT in the management of squamous cell carcinoma of the head and neck and thyroid carcinoma.
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2007 J48 Suppl 1:58S-67S.Clinical role of 18F-FDG PET/CT in the management of squamous cell carcinoma of the head and neck and thyroid carcinoma.1, , , , , , .1Division of Nuclear Medicine, Department of Radiology, Stanford University Medical Center, Stanford, California 94305, USA. aquon@stanford.eduAbstract18F-FDG PET/CT has rapidly become a widely used imaging modality for evaluating a variety of malignancies, including squamous cell carcinoma of the head and neck and thyroid cancer. Using both published data and the multidisciplinary experience at our institution, we provide a practical set of guidelines and algorithms for the use of 18F-FDG PET/CT in the evaluation and management of head and neck cancer and thyroid cancer.PMID:
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External link. Please review our .本人差3个月就14岁了，身高1米56，男的，体重48公斤，肚脐眼的话腿长95cm,那么我的腿算长吗，_百度知道
本人差3个月就14岁了，身高1米56，男的，体重48公斤，肚脐眼的话腿长95cm,那么我的腿算长吗，
这个暑假想长高5公分，长的到吗我马上放暑假，那我什么时候吃鸡公好啊，那我暑假做什么运动好，差3个月就14岁了，男的
我大爷爷1米80
我有更好的答案
你好，身高主要由两方面因素决定，先天因素（占70%）和后天因素（占30%），先天因素就是父母的遗传，有一个遗传预测身高公式，是父母的平均身高*1.08=男孩成年时的预计身高，如果想长得高更，就要充分利用后天因素，要补钙、多晒太阳、多做室外运动、每天吊单杠，能刺激骨骼增长。还有就是要保证充足的睡眠。具体一点说就是饮食上要多吃芝麻、黄花菜、荠菜、海带、田螺、虾皮、豆腐、牛奶。骨类汤有助于钙的吸收，晒太阳也有助于钙的吸收。还要多吃新鲜的蔬菜水果，少吃糖、巧克力、甜蛋糕，这些阻碍钙代谢。适合长高做的运动有，.摸高、爬杆或爬绳梯锻炼、引体向上、交叉伸展、跳绳、跳皮筋、踢毽子、单杆悬垂、游泳、打篮球。这些训练都会增加关节、韧带的柔韧性，有助于身高发育。像举重、杠铃、铅球、铁饼等...
那我天天最少摸高多少下，谢谢大家，一天嗮多少分钟太阳
这个没有硬性规定，根据你自身条件而定，晒太阳最好每天一小时以上。一般来讲付出的与得到的成正比。祝你愉快！
去干农活，越晒越健康，让骨骼进行光合作用促进骨骼生长
你的身高很大程度上可以参考你爸爸的身高。
我爸爸才1米69，妈妈才1米55额
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出门在外也不愁[The expression of p53, p16, cyclin D1 in esophageal squamous cell carcinoma and esophageal dysplasia].
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):269-76.[The expression of p53, p16, cyclin D1 in esophageal squamous cell carcinoma and esophageal dysplasia]. [Article in Korean]1, , , , , , , , , , , , , , , .1Institute for Digestive Research, Department of Internal Medicine, Soon Chun Hyang University College of Medicine, Wonmi-Gu, Bucheon, Korea.AbstractBACKGROUNDS/AIMS: p53 is known to play a central role in sensing and signaling for the growth arrest and apoptosis in cells with DNA damage. Mutation of p53 is a frequent event in esophageal squamous cell carcinoma (ESCC). p16 protein binds to cyclin dependent kinase 4 (CDK4) inhibiting the ability of CDK4 to interact with cyclin D1, and stimulates the passage through the G1 phase of cell cycle. We observed the expression patterns and frequencies of p53, p16, and cyclin D1 in esophageal dysplasia and in esophageal squamous cell carcinomas.METHODS: In 15 patients of ESCC, 5 patients of esophageal dysplasia and 5 volunteers with normal esophagus, tissue specimens were taken from esophageal lesions during the operation or endoscopic examination. We used specific monoclonal antibodies for p53 protein, p16(INK4 ) protein and cyclin D1. Immunoreactivity was scored.RESULTS: Mean age of all groups was 66 years old (range 47-93) and men to women ratio was 19:1. p53 mutation was observed in 87% (13/15) of ESCC, in 80% (4/5) of esophageal dysplasia, in 0% (0/5) of normal mucosa (p=0.001). p16 expression was seen in 40% (2/5) of esophageal dysplasia, 27% (4/15) of ESCC and 100% (5/5) of normal mucosa (p=0.016). Cyclin D1 expression was not significantly different among 20% (1/5) of esophageal dysplasia, 53% (8/15) of ESCC and 20% (1/5) of normal mucosa. Either the expression of p53 mutation or the loss of p16 occurred in 80% (4/5) of esophageal dysplasia and in 93% (14/15) of ESCC.CONCLUSIONS: The expression of p53 mutation and the loss of p16 might play a central role in the pathogenesis of esophageal squamous cell carcinoma (ESCC), and contribute to the development of precancerous lesion such as dysplasia. In addition, there is a possibility that the mutations of p53 and p16 silencing would be the early events in ESCC development.PMID:
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External link. Please review our .女生14岁，身高168，体重62KG，三围98/69/96，罩杯E，腿长112cm，身材怎么样，是不是胖了？_百度知道
女生14岁，身高168，体重62KG，三围98/69/96，罩杯E，腿长112cm，身材怎么样，是不是胖了？
了的话怎么减肥..？还有胸太大了能不能减小...
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这点有优势也有劣势才14岁.胸确实比一般的人大.....,但是你不算胖的保持好身材
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从70E的胸围来看，你是很苗条而ru房很丰满的姑娘再加上你现在是14岁你的胸围是70E，也就是34E的罩杯，真是让多少男人愿意出生入死啊
你的身材已经和你的身体不协调了太可惜了
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出门在外也不愁Metabolism of phytol to phytanic acid in the mouse, and the role of PPARalpha in its regulation.
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):77-85. Epub
2006 Oct 2.Metabolism of phytol to phytanic acid in the mouse, and the role of PPARalpha in its regulation.1, , , , , , .1Laboratory of Genetic Metabolic Diseases, Department of Clinical Chemistry, Emma's Children's Hospital, Academic Medical Center, University of Amsterdam, 1105 AZ Amsterdam, The Netherlands.AbstractPhytol, a branched-chain fatty alcohol, is the naturally occurring precursor of phytanic and pristanic acid, branched-chain fatty acids that are both ligands for the nuclear hormone receptor peroxisome proliferator-activated receptor alpha (PPARalpha). To investigate the metabolism of phytol and the role of PPARalpha in its regulation, wild-type and PPARalpha knockout (PPARalpha-/-) mice were fed a phytol-enriched diet or, for comparison, a diet enriched with Wy-14,643, a synthetic PPARalpha agonist. After the phytol-enriched diet, phytol could only be detected in small intestine, the site of uptake, and liver. Upon longer duration of the diet, the level of the (E)-isomer of phytol increased significantly in the liver of PPARalpha-/- mice compared with wild-type mice. Activity measurements of the enzymes involved in phytol metabolism showed that treatment with a PPARalpha agonist resulted in a PPARalpha-dependent induction of at least two steps of the phytol degradation pathway in liver. Furthermore, the enzymes involved showed a higher activity toward the (E)-isomer than the (Z)-isomer of their respective substrates, indicating a stereospecificity toward the metabolism of (E)-phytol. In conclusion, the results described here show that the conversion of phytol to phytanic acid is regulated via PPARalpha and is specific for the breakdown of (E)-phytol.PMID:
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